Fig. 2From: Elk1 enhances inflammatory cell infiltration and exacerbates acute lung injury/acute respiratory distress syndrome by suppressing Fcgr2b transcriptionTh17 cell infiltration in ARDS rats is reduced by overexpressing Fcgr2b. Notes A, Immunohistochemistry to examine Fcgr2b expression in lung tissues of ARDS rats; B, immunohistochemistry to examine Fcgr2b expression in lung tissues of AAV-Fcgr2b-treated ARDS rats; C, immunohistochemistry to measure RORγt expression in lung tissues of each group of rats; D, Flow cytometry to determine the cell counts of Macrophage, Neutrophils, Lymphocytes and Th17 cells in BALF of each group of rats; E, ELISA to detect IL-17 and IL-6 expression in BALF of each group of rats. F, Photographs showing Evans blue accumulation in lung tissues (n = 6/group). Differences between two groups were compared using the t-test (A-B), and differences among multiple groups were compared using one-way or two-way analysis of variance with Tukey’s post-hoc test (C-E). AAV, adeno-associated virus; ARDS, acute respiratory distress syndrome; BALF, bronchoalveolar lavage fluidBack to article page