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Fig. 5 | Molecular Medicine

Fig. 5

From: Calcitriol confers neuroprotective effects in traumatic brain injury by activating Nrf2 signaling through an autophagy-mediated mechanism

Fig. 5

Calcitriol (0.5 µg/kg) treatment ameliorated TBI-induced oxidative stress. a Malondialdehyde (MDA) status. b Catalase (CAT) activity. c Superoxide dismutase (SOD) activity. d Glutathione (GSH) status. e Representative images of immunofluorescence assays of dihydroethidium (DHE) (Bar = 50 µm). f Statistical graphs of DHE-positive cells in different groups. Data are presented as means ± SEM (n = 5). **P < 0.01 versus the indicated groups. (Sham: sham-operated group; TBI: TBI model group; Calcitriol: TBI + calcitriol treatment group)

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Molecular Medicine

ISSN: 1528-3658