Fig. 2From: Gut bacterial metabolite Urolithin A inhibits myocardial fibrosis through activation of Nrf2 pathway in vitro and in vivoUA treatment repressed TGF-β1-induced transformation of cardiac fibroblasts to myofibroblasts. A The mRNA and B protein levels of vimentin, DDR2, α-SMA and tensin in CFs after indicated treatments. β-actin was used as normalized control. N = 3, *P < 0.05, **P < 0.01, ***P < 0.001Back to article page