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Fig. 3 | Molecular Medicine

Fig. 3

From: c-Jun-mediated miR-19b expression induces endothelial barrier dysfunction in an in vitro model of hemorrhagic shock

Fig. 3

Overexpression of c-Jun enhances hypoxia-induced pri-miR-19b expression. HEK293T cells were transfected with pMIEG3-c-Jun expression vector (cJun-ov) or pMIEG3 empty vector control (Ctrl) then exposed to H/R or normoxia (N). The expression of c-Jun-GFP was examined under inverted fluorescent microscopy and images taken (A) and was analyzed by Western blotting (B). Quantitative real-time PCR for pri-miR-19b was also shown (C)

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