Skip to main content
Fig. 3 | Molecular Medicine

Fig. 3

From: DYNLT1 promotes mitochondrial metabolism to fuel breast cancer development by inhibiting ubiquitination degradation of VDAC1

Fig. 3

High expression of DYNLT1 promotes the proliferation, clone formation, and migration of breast cancer

A-B RT-qPCR and Western blot showed the successful overexpression of DYNLT1 in MCF-7 (A) or MDA-MB-468 (B) breast cancer cells. C-D The proliferation after overexpression of DYNLT1 in MCF-7 (C) or MDA-MB-468 (D) breast cancer cells. E-F The ability of clone formation after overexpression of DYNLT1 in MCF-7 (E) or MDA-MB-468 (F) breast cancer cells. The graph on the right shows the statistics of the number of clones. G-H The migration after overexpression of DYNLT1 in MCF-7 (G) or MDA-MB-468 (H) breast cancer cells. *, p < 0.05; **, p < 0.01; ****, p < 0.0001. All experiments were performed at least three replicates

Back to article page

Molecular Medicine

ISSN: 1528-3658