Fig. 8From: USP38 exacerbates atrial inflammation, fibrosis, and susceptibility to atrial fibrillation after myocardial infarction in miceUSP38 promotes post-MI inflammation by regulating the activation of the TAK1/NF- k B signaling pathway. (A) Representative Western blots of the protein levels (P-TAK1/TAK1, P-P65/P65, P-IκBα/ IκBα) from USP38-CKO mice after MI. (B-D) Statistical analysis of the protein levels in the four groups (n = 4). (E) Representative Western blots of the protein levels from USP38-TG mice after MI. (F-H) Statistical analysis of the protein levels in the four groups (n = 4), *P < 0.05 vs. Sham group, #P < 0.05 vs. Flox-MI or NTG-MIBack to article page