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Table 2 The association between SNP rs11066001 in BRAP and carotid atherosclerosis.

From: BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

Phenotype

 

Genotype

 

Statistics (recessive model)

Carotid plaque

AA

AG

GG

Logistic regressiona

Absence

563 (50.6%)

472 (42.4%)

77 (6.9%)

Reference

Presence

278 (48.9%)

235 (41.3%)

56 (9.8%)

OR = 1.58 (1.08–2.34);

    

P = 0.021

Carotid IMT

    

Continuous IMT data

AA (n = 700)

AG (n = 571)

GG (n = 100)

Multivariate regressiona

CCA, mean ± SD, mm

0.62 ± 0.14

0.63 ± 0.14

0.65 ± 0.16

P = 0.089

Bif, mean ± SD, mm

0.65 ± 0.13

0.66 ± 0.13

0.66 ± 0.13

P = 0.732

ICA, mean ± SD, mm

0.51 ± 0.10

0.51 ± 0.09

0.51 ± 0.10

P = 0.834

Dichotomized IMT, ≥mean + 1 SD versus < mean + 1 SD

   

Logistic regressiona

CCA

46.0%

43.1%

10.9%

OR = 2.25 (1.30–3.89),

 

versus 52.0%

versus 41.4%

versus 6.6%

P = 0.004

Bif

49.0%

43.7%

7.3%

OR = 1.12 (0.61–2.03),

 

versus 51.5%

versus 41.2%

versus 7.3%

P = 0.720

ICA

54.5%

39.6%

5.9%

OR = 0.77 (0.40–1.50);

 

versus 50.5%

versus 42.0%

versus 7.5%

P = 0.445

  1. aP value obtained from multivariate regression analysis with adjustment for age, sex, hypertension, diabetes, hyperlipidemia, BMI and smoking. Only covariates with P< 0.05 were kept in the regression models.