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Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

A Research Article was published on 20 March 2014

The Editors-in-Chief would like to alert readers that this article (Sitapara et al. 2014) is part of an investigation being conducted by the journal following the conclusions of an institutional enquiry at the University of Liverpool with respect to the quantitative mass spectrometry-generated results regarding acetylated and redox-modified HMGB1.

Expression of Concern to: Mol Med

https://doi.org/10.2119/molmed.2013.00086

The Editors-in-Chief would like to alert readers that this article (Sitapara et al. 2014) is part of an investigation being conducted by the journal following the conclusions of an institutional enquiry at the University of Liverpool with respect to the quantitative mass spectrometry-generated results regarding acetylated and redox-modified HMGB1. Appropriate editorial action will be taken once the investigation is concluded.

Lokesh Sharma, Charles R. Ashby Jr., Saamir Gorasiya, Huan Yang, Ravikumar A. Sitapara, Vivek S. Patel, and Lin L. Mantell agree to this editorial expression of concern. We were unable to contact author Michelle Zur. Daniel J. Antoine has not responded to any correspondence from the editor/publisher about this editorial expression of concern.

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  1. Sitapara, et al. The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function. Mol Med. 2014;20:238–47 https://molmed.biomedcentral.com/articles/10.2119/molmed.2013.00086.

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Correspondence to Lin L. Mantell.

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Sitapara, R.A., Antoine, D.J., Sharma, L. et al. Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function. Mol Med 26, 16 (2020). https://doi.org/10.1186/s10020-020-0143-9

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