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Correction to: METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α‑klotho
Molecular Medicine volume 28, Article number: 8 (2022)
The Original Article was published on 09 September 2021
Correction to: Mol Med (2021) 27:106 https://doi.org/10.1186/s10020-021-00365-5
Following publication of the original article (Li et al. 2021), the authors identified an error in Fig. 2. The correct Fig. 2 is given in this erratum.
METTL14 promoted high glucose-induced glomerular endothelial cell injury. A Expression of METTL14 was examined by qRT-PCR after transfected with Vector or MELLT14 plasmid. **P < 0.01 vs. Vector. B Expression of METTL14 was examined by qRT-PCR after transfected with si-ctrl or MELLT14 siRNA. **P < 0.01 vs. si-Ctrl. C–E The levels of ROS, TNF-α and IL-6 were detected by ELISA. F Cell proliferation was assessed by CCK-8. G, H Cell apoptosis was analysed by TUNEL staining, Scale bar: 50 μm. *P < 0.05, **P < 0.01 vs. NG; #P < 0.05, ##P < 0.01 vs. HG + Vector; &P < 0.05, &&P < 0.01 vs. HG + si-Ctrl. Data are presented as the mean ± SD (n = 3)
The original article has been corrected.
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Li M, Deng L, Xu G. METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α-klotho. Mol Med. 2021;27:106. https://doi.org/10.1186/s10020-021-00365-5.
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Li, M., Deng, L. & Xu, G. Correction to: METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α‑klotho. Mol Med 28, 8 (2022). https://doi.org/10.1186/s10020-022-00437-0
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DOI: https://doi.org/10.1186/s10020-022-00437-0